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Human evidence record ยท Omberacetam (Noopept)

Clinical and EEG characteristics of Noopept in mild cognitive impairment of vascular or traumatic origin

Noopept was associated with increased alpha/beta and reduced delta EEG power, interpreted as nonspecific activation/anxiolytic-type changes.

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Human evidence record

What this study tells us

This is human clinical evidence. Its strength depends on the study design, sample size, comparator, population, duration and the outcome that was actually measured.

Who was studied?

Patients with cognitive disturbances after vascular or traumatic brain disease

How many?

n=Not stated in abstract

What happened?

Noopept was associated with increased alpha/beta and reduced delta EEG power, interpreted as nonspecific activation/anxiolytic-type changes.

What stops us overclaiming?

Russian-language study; no placebo-controlled blinded design established from the abstract.

Need help with terms such as randomized, placebo, endpoint or sample size? Open the glossary โ†’

Key result

Noopept was associated with increased alpha/beta and reduced delta EEG power, interpreted as nonspecific activation/anxiolytic-type changes.

Study details

Study designClinical study with EEG outcomes
PopulationPatients with cognitive disturbances after vascular or traumatic brain disease
Sample sizeNot stated in abstract
RouteOral
Regimen / exposureClinical Noopept treatment; exact regimen not reproduced in the abstract
DurationClinical course
ComparatorNo placebo control described in abstract
Primary endpoint / questionEEG rhythm changes and cognitive/asthenic clinical context
Safety resultContemporary safety characterization was limited.
Key limitationsRussian-language study; no placebo-controlled blinded design established from the abstract.
Registry / identifierPMID 19008801